ConditionBrain & nerves
Vertigo: Types, Diagnosis and Treatment
Vertigo is an illusion of movement, usually spinning, caused by a mismatch in the balance (vestibular) system. Doctors classify it as peripheral, arising from the inner ear or vestibular nerve, which accounts for most cases, or central, arising from the brainstem or cerebellum, as in stroke, vestibular migraine or multiple sclerosis. The distinction guides everything from urgency to treatment. Most peripheral vertigo is curable or improves with repositioning maneuvers and vestibular rehabilitation.
Also called: Vestibular vertigo, spinning dizziness, balance disorder
At a glance
- What it is
- A false sense of motion from a disturbance in the inner ear, vestibular nerve or brain balance centers.
- Peripheral (most cases)
- BPPV, vestibular neuritis, labyrinthitis, Ménière's disease, perilymph fistula.
- Central
- Posterior circulation stroke or TIA, vestibular migraine, multiple sclerosis, tumors.
- Key tests
- Dix-Hallpike, HINTS exam, hearing tests, vestibular tests and MRI when central cause is possible.
- Treatment
- Repositioning maneuvers, short-term vestibular suppressants, vestibular rehabilitation, and treating the cause.
When to get help
Call 911 or go to the ER
- Vertigo with double vision, slurred speech, facial weakness, limb weakness or numbness, or trouble swallowing
- Vertigo with inability to stand or walk unaided
- Sudden vertigo with a severe headache or neck pain
See a doctor within a day or two
- New continuous vertigo lasting hours with vomiting, especially in someone over 50 or with vascular risk factors
- Vertigo with sudden one-sided hearing loss
Book a routine appointment
- Recurrent brief positional vertigo
- Recurrent episodes with ear fullness, tinnitus or fluctuating hearing
- Persistent imbalance or motion sensitivity after an attack
Usually safe to manage at home
- Home repositioning exercises for confirmed BPPV, as taught by a clinician
How the balance system works
The inner ear contains three semicircular canals, which detect head rotation, and two otolith organs (utricle and saccule), which detect gravity and straight-line movement. Signals travel along the vestibular nerve to the brainstem and cerebellum, where they are combined with information from the eyes and from joints and muscles. Vertigo results when one side sends abnormal signals, or when the brain cannot process them correctly.
Peripheral vs central vertigo
| Feature | Peripheral | Central |
|---|---|---|
| Source | Inner ear or vestibular nerve | Brainstem or cerebellum |
| Share of cases | Most | Minority |
| Nausea and vomiting | Often severe | Variable, can be mild |
| Hearing loss or tinnitus | Can occur | Usually absent |
| Nystagmus | One direction, horizontal-torsional, suppressed by fixing the gaze | May change direction, vertical, not suppressed |
| Walking | Unsteady but can walk | Often cannot walk unaided |
| Other neurological signs | None | Often: double vision, slurred speech, weakness, numbness |
| Head impulse test | Abnormal | Usually normal |
Peripheral causes
Benign paroxysmal positional vertigo
BPPV is the most common vestibular disorder. Detached otoconia (calcium carbonate crystals) move into a semicircular canal, most often the posterior canal. Brief vertigo occurs with head position changes. Lifetime prevalence is around 2 to 3% and it is more common in older adults, women, after head injury and with low vitamin D.
Vestibular neuritis and labyrinthitis
Viral inflammation of the vestibular nerve (vestibular neuritis) causes acute continuous vertigo over days. When hearing is involved, it is labyrinthitis. Recovery happens through central compensation over weeks.
Ménière's disease
Ménière's disease is linked to excess fluid (endolymphatic hydrops) in the inner ear. Diagnosis requires two or more spontaneous attacks lasting 20 minutes to 12 hours, low-to-mid frequency hearing loss on testing, and fluctuating ear symptoms.
Other peripheral causes
- perilymph fistula after barotrauma or surgery
- superior canal dehiscence
- vestibulotoxic drugs, such as gentamicin
- acoustic neuroma (vestibular schwannoma), usually causing hearing loss more than vertigo
- herpes zoster oticus (Ramsay Hunt syndrome)
Central causes
Posterior circulation stroke and TIA
Strokes in the vertebrobasilar territory can cause vertigo, sometimes as the only symptom. Studies of patients with acute continuous vertigo show the bedside HINTS exam, done by trained clinicians, is more sensitive than early CT for detecting stroke. MRI is the imaging test of choice. See stroke.
Vestibular migraine
Vestibular migraine is now recognized as one of the most common causes of recurrent vertigo. Diagnosis requires a migraine history, recurrent vestibular episodes of 5 minutes to 72 hours, and migraine features with at least half the episodes.
Multiple sclerosis
Demyelinating plaques in the brainstem can cause vertigo, often with other neurological symptoms. See multiple sclerosis.
Other central causes
Cerebellar tumors, Chiari malformation, and some medication or alcohol toxicity.
Diagnosis
History
The key questions concern timing and triggers:
- Triggered episodic vertigo (seconds, with head movement): BPPV
- Spontaneous episodic vertigo (minutes to hours): Ménière's, vestibular migraine, TIA
- Acute continuous vertigo (days): vestibular neuritis, labyrinthitis, stroke
- Chronic dizziness (months): persistent postural-perceptual dizziness (PPPD), uncompensated vestibular loss, bilateral vestibular loss
Bedside tests
- Dix-Hallpike maneuver: diagnoses posterior canal BPPV
- Supine roll test: diagnoses horizontal canal BPPV
- HINTS exam: Head Impulse, Nystagmus pattern and Test of Skew, used in acute continuous vertigo to separate vestibular neuritis from stroke
- Gait and coordination testing
Further tests
- Audiometry: hearing loss patterns
- Videonystagmography (VNG), caloric testing, video head impulse test (vHIT), VEMPs: measure vestibular function on each side
- MRI of the brain and internal auditory canals: for suspected central cause, acoustic neuroma or asymmetrical hearing loss
Treatment
Canalith repositioning for BPPV
The Epley and Semont maneuvers move crystals back into the utricle. AAO-HNS guidance recommends them as first-line treatment, with success rates of about 80% after one to three maneuvers. Post-maneuver restrictions are generally not needed. Recurrence is common, about 15% per year.
Vestibular suppressants
Antihistamines such as meclizine, antiemetics and benzodiazepines can ease severe acute vertigo, but should be limited to a few days. Prolonged use delays central compensation.
Vestibular rehabilitation therapy
Exercises that include gaze stabilization, habituation and balance training help the brain adapt. It is effective for vestibular neuritis, uncompensated vestibular loss, and chronic dizziness.
Cause-specific treatment
- Vestibular neuritis: early corticosteroids may aid recovery of nerve function, though evidence for long-term benefit is mixed
- Ménière's disease: salt restriction, diuretics, betahistine in some countries, intratympanic steroids or gentamicin, and surgery in refractory cases
- Vestibular migraine: lifestyle and trigger management, migraine preventives
- Stroke: acute stroke treatment and secondary prevention
- PPPD: vestibular rehabilitation, CBT and SSRIs
Complications
- falls and injuries, particularly in older adults
- anxiety and avoidance behavior
- chronic dizziness (PPPD) after an acute vestibular event
- dehydration from vomiting
Outlook
The outlook for peripheral vertigo is generally good. BPPV usually resolves with treatment, though it can recur. Most people recover from vestibular neuritis within weeks to months, especially with rehabilitation. Ménière's disease is chronic, but attacks can be controlled for most. Central vertigo outcomes depend on the cause.
For a practical guide to vertigo symptoms and when to seek help, see vertigo symptoms.
Frequently asked questions
What is the difference between peripheral and central vertigo?
Peripheral vertigo comes from the inner ear or vestibular nerve and is the most common type, usually with severe nausea and sometimes hearing changes. Central vertigo comes from the brain, often with other neurological signs such as double vision, slurred speech or inability to walk.
What is the most common type of vertigo?
Benign paroxysmal positional vertigo (BPPV) is the most common. It causes brief episodes of spinning triggered by head movements such as rolling over in bed, caused by loose crystals in the inner ear.
How is vertigo diagnosed?
Diagnosis relies on the history of timing and triggers plus bedside tests such as the Dix-Hallpike maneuver and the HINTS exam. Hearing tests and vestibular function tests help, and MRI is used when a brain cause is possible.
What is the best treatment for vertigo?
It depends on the cause. BPPV is best treated with repositioning maneuvers. Vestibular neuritis and long-term imbalance improve with vestibular rehabilitation. Ménière's disease and vestibular migraine have specific treatments. Medicines only help briefly in acute attacks.
Can vertigo be cured?
Many causes can. BPPV is usually cured with repositioning maneuvers, though it may recur. Vestibular neuritis typically resolves as the brain compensates. Ménière's disease cannot be cured but can be controlled. Treatment of the underlying cause is key.
Sources
This guide was written from current patient and clinical guidance, including:
- American Academy of Otolaryngology-Head and Neck Surgery (AAO-HNS): Clinical practice guideline: Benign paroxysmal positional vertigo (update)
- Bárány Society: International Classification of Vestibular Disorders
- American Academy of Neurology: Practice parameter on BPPV
- National Institute on Deafness and Other Communication Disorders (NIDCD): Balance disorders
- Vestibular Disorders Association (VeDA): Types of vestibular disorders